Mini-Gastric Bypass

The Mini (Sleeve) Gastric Bypass Surgery


Short, Simple, Effective

Short, Simple, Effective Weight Loss Surgery

Post Gastrectomy

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POST GASTRECTOMY SYNDROMES

bulletThese may be due to either disruption of normal physiology of GI motility or due to nutritional defects occurring due to gastrectomy.
bulletChanges after gastrectomy :
  1. Decreased acid
  2. Decreased pepsin
  3. Decreased Intrinsic Factor
  4. Decreased pancreatic enzymes
  5. Decreased mixing of food with acid, pepsin, and bile.
  6. Decreased absorption of proteins, calcium, Vitamin D & B, Fe, fat
  7. Increased absorption of glucose (RAPID absorption).
  8. Increased intestinal motility
  9. Creation of a "blind loop" i.e. afferent loop.
bulletPostgastrectomy syndrome classification :
bulletPost Meal syndromes - these start within 1-2 weeks after the operation.
bulletEarly Post Meal syndrome (Syn. Early dumping)
bulletLate Post Meal syndrome (Syn. Late dumping OR hypoglycemia)
bulletBilious vomiting
bulletNutritional syndromes :
bulletWeight loss
bulletSteatorrhea
bulletDiarrhea
bulletAnemia - either Fe deficiency or megaloblastic
bulletVit B deficiency
bulletCalcium deficiency
bulletGross malabsorption syndrome
bulletPost Meal SYNDROMES
bulletClassified as early & late according to the onset of symptoms after food. Both arise at about the same time span after operation.
bulletBoth consist of attacks lasting about 30-40 min.
bulletEarly Post Meal syndrome -
bulletSeen in most patients in convalescence but persists in 5-12%.
bulletCommon after Billroth II gastrectomy as compared to Billroth I.
bulletSymptoms start immediately after meals.
bulletPathogenesis - primary defect of CHO metabolism. Rapid glucose absorption - hyperglycemia - no further absorption of glucose from gut - osmotic drawing in of water into lumen - distention of GIT due to fluid - increased intestinal motility & volume depletion from circulation.
bulletC/F - Epigastric fullness, Sensation of warmth, sweating, tachycardia, vomiting or diarrhea. Aggravated by wet foods, foods with CHOs, and large amounts of food. Relief on lying down.
bulletTreatment - often subsides on its own in the course of time.
bulletSmall, frequent, dry meals
bulletAvoid milk & CHO rich foods
bulletCodeine to reduce intestinal hypermotility
bulletMaintain adequate Hb levels
bulletIf does not subside - convert Billroth II into Billroth I OR slow transit time by interposition of 10 cm loop of anti-peristaltic jejunum.
bulletLate Post Meal syndrome :
bulletSymptoms start 2 to 2 ½ hours after meals.
bulletOccurs after any type of gastrectomy (not only Billroth II)
bulletThe pathology is a hypoglycemia. Initial rapid absorption of glucose - hyperglycemia - increased Insulin - fall in BSL to about 50 mg%.
bulletC/F - epigastric emptiness, faintness, tremors, nausea
bulletAggravated by exercise, and relief by more food or glucose.
bulletTreatment - Small frequent meals, preferably dry. On symptoms glucose can be taken.
bulletBilious vomiting :
bulletOccurs in 10-15% of patients, may be associated with dumping.
bulletMay be caused due to probable transient obstruction of the afferent loop in Billroth II gastrectomy.
bulletFood eaten passes on into efferent loop without any bile.
bulletOnce food passes on, afferent loop (containing bile) empties into stomach & there is bilious vomiting.
bulletTreatment - may be relieved by relief of obstruction of afferent loop or Billroth II may be converted into Billroth I type.
bulletNUTRITIONAL SYNDROMES
bulletWeight loss :
bulletAlways in total gastrectomy, frequently in Billroth II partial gastrectomy & rarely in Billroth I.
bulletAlways reactivation of TB due to malnutrition should be excluded.
bulletOccurs due to reduced food intake & malabsorption.
bulletMay be beneficial - post-gastrectomy pts have less incidence of IHD.
bulletSteatorrhea :
bulletPoor mixing of food with digestive enzymes
bulletFormation of a blind loop syndrome.
bulletDiarrhea :
bulletMost post-gastrectomy patients have improved motility but 5% have diarrhea.
bulletMay be due to steatorrhea or intestinal hypermotility (dumping).
bulletTreated with Loperamide or diphenoxylate-atropine (Lomotil)
bulletAnemia :
bulletMay be Fe def or megaloblastic anemia.
bulletFe def anemia common when duodenum is by-passed e.g. Billroth II. Treated by excluding occult blood loss and FSFA tablets.
bulletMegaloblastic anemia - common with total gastrectomy. May be due to atrophic gastritis or part of the Gross malabsorption syndrome. Managed by measuring Vit B12 levels and treating with Inj Cyanocobalamine 1000 mcg IM weekly till serum levels normalize then given monthly. Post gastrectomy prophylaxis is usually 1000 micrograms Inj IM once a year.
bulletCalcium deficiency :
bulletdue to associated steatorrhea
bulletdue to asso Vit D deficiency
bulletdue to less acidic pH which causes less absorption per se.
bulletGross malabsorption syndrome :
bulletCombination of all of above deficiencies.
bulletTreated by conversion of Billroth II to Billroth I.
bulletSmall stomach syndrome :
bulletCommon but mild condition - presents as early satiety, epigastric fullness, with consequent reduced food intake - with weight loss & other nutritional deficiencies.
bulletTreated with high energy meals with small volumes.
bulletOTHER COMPLICATIONS OF GASTRIC SURGERY -
bulletEarly -
bulletHemorrhage from anastomotic line.
bulletStomal obstruction - may be initially due to tissue oedema immediately after surgery or later due to jejuno-gastric intussception.
bulletDuodenal "blow-out" - forms duodeno-cutaneous fistula.
bulletParalytic ileus - treated with "suck + drip".
bulletLater -
bulletRecurrent ulceration.
bulletPost gastrectomy syndromes
bulletPost vagotomy syndromes
bulletResidual abscess - may later lead to peritonitis or fistula.
bulletRecurrent neoplastic ulcer.
bulletAnemia
bulletOsteoporosis
bulletGastro-jejuno-colic fistula.
 

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Contact Information: -Telephones: *** CLOS West: 702-456-4643; Trish Lanman 702-376-3446, Sandy Brubaker 702-376-3647; Jennifer Brubaker 702-376-9339, Dr. Rutledge 702-215-9550 *** CLOS Florida: Flo Ballengee 863-899-3463 Dr. Cesare Peraglie 407-922-3424


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Email: Dr. Rutledge DrR@clos.net, *** CLOS West: Trish Lanman Trish@clos.net, Sandy Brubaker SandyB@clos.net Dr. Rutledge DrR@clos.net *** CLOS Florida: Flo Ballengee flo@clos.net, Dr. Peraglie drp@clos.net


Addresses:
Address: *** CLOS West Office: Dr Robert Rutledge / CELOS, 98 E Lake Mead Parkway Suite 302, Henderson NV 89015, Office 702-456-4643, Office fax: 702-456-1173, Contacts: Trish Lanman 702-376-3446 Trish@clos.net, Sandy Brubaker 702-376-3647 SandyB@clos.net, Jennifer Brubaker 702-376-9339 Jen@clos.net, Dr. Rutledge 702-215-9550 Drr@clos.net *** CLOS Florida: 40124 Highway 27, Suite 203, Davenport, FL 33837, Flo Ballengee 863-899-3463, Flo@clos.net, Dr. Peraglie 407-922-3424 drp@clos.net


Warning: Gastric Bypass Surgery is a MAJOR surgical procedure. It can be associated with significant risks and complications, up to and including death. Weight loss surgery is a rapidly developing area of medicine. Bariatric surgery is filled with controversy. It is very important to take a careful and deliberate approach to considering surgery for the treatment of obesity.  

Disclaimer Notice:-Information on this web site is provided for informational purposes only.
-It is imperative that you consult your own physician regarding the applicability of any opinions or recommendations with respect to your symptoms or medical condition.
-Contact with this web site or Dr. Rutledge over the web site does not constitute a doctor patient relationship and for good quality medical care you must obtain advice and consultation form your own local physician.
-This site is intended as a resource for references on the treatment of obesity for health care professionals and educated consumers.
-The authors and editors have used sources believed to be reliable in their efforts to provide information that is complete and generally in accord with the standards accepted at the time of publication.
-Medical knowledge changes rapidly. In view of the possibility of human error or changes in medical science, neither the authors nor the editors nor any other party who has been involved in the preparation or publication of this work warrants that the information contained herein is in every respect accurate or complete, and they are not responsible for any errors or omissions or for the results obtained from the use of such information.
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